Speaking to Baylor University, study co-author Dr Benjamin Deneen said that increasing levels of the protein, which “regulates astrocyte functions during ageing”, helped to clear plaques “from the ...
A bacteria best known for causing stomach ulcers could lead to a new strategy in the fight against Alzheimer’s disease ...
A loss of brain volume associated with new immunotherapies for Alzheimer's disease may be caused by the removal of amyloid plaques, rather than the loss of neurons or brain tissue, finds a study led ...
A new study identifies a biological process that empowers specific brain cells to eliminate harmful proteins associated with ...
Researchers discovered that raising the protein Sox9 can help the brain’s astrocytes clear out toxic plaque buildup linked to Alzheimer’s. In mouse models that already showed memory problems, ...
Lecanemab, sold under the name Leqembi, is a monoclonal antibody therapy for Alzheimer's disease that clears toxic amyloid plaques and delays cognitive decline. Researchers from VIB and KU Leuven have ...
A group from Osaka Metropolitan University in Japan, led by graduate student Tatsushi Oura and Dr. Hiroyuki Tatekawa, found that treatment using the drug lecanemab to remove amyloid plaques in the ...
An experimental drug appears to reduce the risk of Alzheimer's-related dementia in people destined to develop the disease in their 30s, 40s or 50s, according to the results of a new study. The ...
Researchers at Baylor College of Medicine have discovered a natural mechanism that clears existing amyloid plaques in the brains of mouse models of Alzheimer’s disease and preserves cognitive function ...
Top neurologists told Newsweek of natural, science-backed ways to protect your brain from aging and disease—before symptoms ...
In a new study, researchers identified a molecule produced by a particular type of brain cell that reversed the cognitive decline seen in both healthy aging and dementia. It provides a deeper ...
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Prion-Like Brain Damage Can Occur Without Infectious Prions, Study Finds
We may have been overestimating the role of a pathological class of misfolded protein in neurodegenerative disease.
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